Download e-book for iPad: Chronic Pain. Pain Management Series by W. Jay Gary

By W. Jay Gary

ISBN-10: 0849330467

ISBN-13: 9780849330469

Delivering a basic method of the knowledge and administration of all varieties of power discomfort, this booklet bargains a transparent and reader-friendly structure that clarifies systems within the prognosis, review, and remedy of the commonest power non-cancer soreness entities. Describing numerous sorts of intractable non-cancer discomfort, together with neuropathic, somatic, and visceral ache, this resource discusses the numerous to be had sorts of remedies, together with opioid and adjunctive soreness drugs, and the secure and correct use of narcotics for treating continual pain.
Analyzing essentially the most common, dear, and debilitating stipulations, this source:
- opens with sections on anatomy, body structure, and the molecular bases of discomfort and progresses to pathophysiology and the analysis of power non-cancer ache entities, together with neuropathic soreness, myofascial soreness syndromes, fibromyalgia, and protracted tension-type headache
- researches quite a few therapy modalities together with opioid and adjunctive ache drugs, mental care, interventional ache medication, and interdisciplinary ache administration therapy utilizing evidence-based drugs principles
- emphasizes the significance of an interdisciplinary remedy group in dealing with sufferers with persistent pain
includes case histories and reviews from households of continual discomfort sufferers

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54. Grotenhermen F. Pharmacology of cannabinoids. Neuro Endocrinol Lett 2004; 25(1–2):14–23. 55. Ibrahim MM, Rude ML, Stagg NJ, et al. CB2 cannabinoid receptor mediation of antinociception. Pain 2006; 122:36–42. 56. Van Der Stelt M, Di Marzo V. Endovanilloids. Putative endogenous ligands of transient receptor potential vanilloid 1 channels. Eur J Biochem 2004; 271(10):1827–1834. 57. Li DP, Chen SR, Pan HL. VR1 receptor activation induces glutamate release and postsynaptic firing in the paraventricular nucleus.

Acta Physiol Hung 1987; 69:343–354. 6. Mense S, Simons DG. Muscle Pain: Understanding its Nature, Diagnosis and Treatment. Baltimore, Md: Lippincott Williams & Wilkins, 2000:30. 7. Ju G, Hokfelt T, Brodin E, et al. Primary sensory neurons of the rat showing calcitonin gene-related peptide immunoreactivity and their relation to substance P, somatostatin-, galanin-, vasoactive intestinal peptide- and cholecystokinin- immunoreactive ganglion cells. Cell Tissue Res 1987; 247:417–431. 34 Chronic Pain 8.

Again, the close relationship of the CNS, the PNS, the autonomous nervous system (ANS), and the musculoskeletal system would anticipate such findings. 32 Chronic Pain CENTRAL SENSITIZATION The receptive fields (RF) of specific dorsal horn neurons are anatomically defined by the spatial projection of their dendrites and the topography of the central terminals of primary afferents. When a dorsal horn neuron fires, the stimulus from the periphery must lie within the receptive field of that neuron—the specific area from which a stimulus depolarizes the dorsal horn neuronal membrane at a strength that is above its action potential activation threshold (39).

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Chronic Pain. Pain Management Series by W. Jay Gary

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