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By Ignacio Martín-Loeches

ISBN-10: 9533078898

ISBN-13: 9789533078892

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100] Belaaouaj A. Neutrophil elastase-mediated killing of bacteria: Lessons from targeted mutagenesis. Microbes and Infection 2002;4:1259 - 1264. [101] Aarbiou J, Verhoosel RM, Van Wetering S, De Boer WI, Van Krieken JH, Litinov SV, Rabe KF, Hiemstra PS. Neutrophil defensins enhance lung epithelial wound closure and mucin gene expression in vitro. Am J Resp Cell Mol Biol 2004;30:193 201. [102] Cepinskas G, Sandig M, Kvietys PR. Paf induced elastase dependent neutrophil transendothelial migration is associated with the mobilisation of elastase to the neutrophil surface and localised to the migrating front.

Reduction of exacerbations by the pde4 inhibitor roflumilast - the importance of defining different subsets of patients with copd. 1186/1465-9921-1112-1118. [173] Larson M. Clinical recognition of n-acetylcysteine in chronic bronchits. Eur Respir Rev 1992;2:5 - 8. [174] Tunek A. Possible mechanisms behind the anti-inflammatory effects of nacetylcysteine: Is metabolism essential? Eur Respir Rev 1992;2:35 - 38. [175] Leuenberger P, Anderhub PJ, Brandli O, et al. Management of chronic obstructive pulmonary disease.

Patients fulfilling these criteria should be assessed carefully to ensure that treatment, where needed, is started promptly. In order to facilitate this, many patients are now being managed in the community with prophylactic antibiotics and oral corticosteroids, as it has been shown that early intervention is associated with better clinical outcomes (162). 2 Treatments for stable disease Most studies of potential treatments used in chronic bronchitis have not differentiated between chronic bronchitis and COPD, and therefore results should be interpreted with caution.

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Bronchitis by Ignacio Martín-Loeches


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